使える単語・表現集
6. 「何が言えるか（ドーン！）」で使える単語・表現
These observations reveal a previously unknown counter-defense strategy employed by pathogens against potential host antimicrobial responses.
Thus, we propose that Agrobacterium has evolved VirD5 to counteract the host-induced degradation of VirF, thereby maximizing infection efficiency.
Thus, our data further suggest that Agrobacterium VirD5 has evolved to function in plant cells, and more specifically, in the plant nucleus.
These results suggest that VirF degradation may be mediated primarily by other E3 ubiquitin ligases, rather than the autocatalytic mechanism.
Together, our data suggest the presence of a host defense mechanism that targets the Agrobacterium virulence factor for proteasome-dependent degradation.
It is tempting to speculate that such plant-specific DNA repair proteins might also play a role in Agrobacterium T-DNA integration.
Thus, we cannot rule out the possibility that the increased T-DNA integration efficiency in mutants lacking NuA4 or SWR1 could be an indirect effect caused by misregulation of as yet unknown genes involved in the integration process.
However, we cannot exclude the possibility that mutations in CAF-1 result in formation of a relatively loose chromatin structure, which may be more accessible to a foreign DNA.
Given that histone acetyltransferases affect Agrobacterium T-DNA integration, it is plausible that histone deacetylases (HDACs) may also play a role in the integration.
An attractive hypothesis is that epigenetic information at chromatins surrounding a DSB may serve as a "landmark" to be recognized by the T-complex.
In an alternative model, histone modifications may help T-DNA "disguise" as a host chromatin that harbors a DSB.
It remains to be investigated whether the T-complex possesses a preferential affinity to any modified histones.
The relatively simple DNA recognition mechanism presented in this work not only sheds a new light on host-pathogen interactions, but also has substantial implications for future genetic engineering technologies, such as plant gene targeting.
Thus, AraNet, the most extensive gene network for plants available to the research community so far, has the potential to further advance plant research and biotechnology.
Although the precise mechanism underlying the induction of the Ubc9 degradation remains to be determined, the interesting discovery presented in this work reveals additional complexity in the host-pathogen molecular arms race.
The novel finding presented in this study not only sheds new light on the crosstalk between the two phytohormone pathways but also suggests that the PEBP proteins may play diverse roles in plant environmental responses.
These observations led to a hypothesis that Ecp6 scavenges chitins from extracellular spaces and prevents the onset of the chitin-triggered plant immunity for efficient infection.
This striking similarity suggests that the temporal control of SPL gene expression by miRNA156 may represent an integral module that is necessary for proper developmental timing in plants.
The discovery presented in this paper provides additional insight into the multifaceted strategy by which viral pathogens suppress the RNA-silencing defense of their plant hosts.
Together, the data presented in this study provide an additional dimension to plant antiviral defense strategy.
These examples illustrate the importance of the defensive role of the UPS in host-pathogen interactions.
Hijacking the host SCF machinery by pathogens makes biological sense because the SCF complex plays a key role in host defense responses in many cases.